机构：[1]Fudan University Shanghai Cancer Center & Institutes of Biomedical Sciences, School of Basic Medical Sciences, Cancer Institutes, Key Laboratory of Breast Cancer in Shanghai, Shanghai Key Laboratory of Radiation Oncology, The Shanghai Key Laboratory of Medical Epigenetics, Shanghai Medical College, Fudan University, Shanghai, China[2]Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China[3]Department of Pathology, Fudan University Shanghai Cancer Center, Institute of Pathology, Fudan University, Shanghai, China[4]Department of Pathology, School of Basic Medical Sciences, Fudan University, Shanghai, China[5]Institutes of Biomedical Sciences, Fudan University, Shanghai, China[6]Hongqiao International Institute of Medicine, Shanghai Tongren Hospital/Faculty of Basic Medicine, Chemical Biology Division of Shanghai Universities E-Institutes, Key Laboratory of Cell Differentiation and Apoptosis of the Chinese Ministry of Education, Shanghai Jiao Tong University School of Medicine, Shanghai, China[7]State Key Laboratory of Medical Neurobiology, Fudan University, Shanghai, China[8]State Key Laboratory of Cell Biology, Shanghai Institute of Biochemistry and Cell Biology, Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, Shanghai, China[9]Present address: Department of Biochemistry and Molecular Cell Biology, Key Laboratory of Cell Differentiation and Apoptosis of National Ministry of Education, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
This work was supported by the National Key R&D
Program of China (no. 2020YFA0803402, 2019YFA0801703 to Q.-Y.L.),
the Natural Science Foundation of China (no. 82121004, 91959202 to Q.-Y.L.; no. 81872240 to M.Y.) and the Innovation Program of Shanghai
Municipal Education Commission (no. 2023ZKD11 to Q.-Y.L.).
第一作者机构：[1]Fudan University Shanghai Cancer Center & Institutes of Biomedical Sciences, School of Basic Medical Sciences, Cancer Institutes, Key Laboratory of Breast Cancer in Shanghai, Shanghai Key Laboratory of Radiation Oncology, The Shanghai Key Laboratory of Medical Epigenetics, Shanghai Medical College, Fudan University, Shanghai, China[2]Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China
共同第一作者：
通讯作者：
通讯机构：[1]Fudan University Shanghai Cancer Center & Institutes of Biomedical Sciences, School of Basic Medical Sciences, Cancer Institutes, Key Laboratory of Breast Cancer in Shanghai, Shanghai Key Laboratory of Radiation Oncology, The Shanghai Key Laboratory of Medical Epigenetics, Shanghai Medical College, Fudan University, Shanghai, China[2]Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China[7]State Key Laboratory of Medical Neurobiology, Fudan University, Shanghai, China
推荐引用方式(GB/T 7714)：
Qian Lin,Li Na,Lu Xiao-Chen,et al.Enhanced BCAT1 activity and BCAA metabolism promotes RhoC activity in cancer progression[J].NATURE METABOLISM.2023,doi:10.1038/s42255-023-00818-7.
APA：
Qian Lin,Li Na,Lu Xiao-Chen,Xu Midie,Liu Ying...&Lei Qun-Ying.(2023).Enhanced BCAT1 activity and BCAA metabolism promotes RhoC activity in cancer progression.NATURE METABOLISM,,
MLA：
Qian Lin,et al."Enhanced BCAT1 activity and BCAA metabolism promotes RhoC activity in cancer progression".NATURE METABOLISM .(2023)
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